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O2⋅− and H2O2-Mediated Disruption of Fe Metabolism Causes the Differential Susceptibility of NSCLC and GBM Cancer Cells to Pharmacological Ascorbate.
RCTs of diet change through to disease outcome are uncommon and the change has usually been addition or removal of only a single food component.
Trials with whole diets through to disease outcome are rare and dietary changes made by individuals are unlikely to be an exact copy of the prescription.
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NOTE: because of the terror attack towards the end of the 2013 Boston Marathon, 20 finisher numbers for said marathon were not included in the same marathon comparison.
As fundamental defects in oxidative metabolism leading to increased steady-state levels of O2⋅− and H2O2 emerge as targetable hallmarks of cancer cells, the current findings support a generalized mechanism for the application of pharmacological ascorbate in cancer therapy.
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